Hypoxia-Induced Deacetylation Is Required for Tetraploid Differentiation in Response to Testicular Ischemia-Reperfusion (IR) Injury

Journal of Andrology, 05/04/2012

The current data suggest that the hypoxia–induced deacetylation may operate as an indispensible defensive mechanism for meiotic differentiation during the ischemic period of ischemia/reperfusion testis, thus pointing out a novel therapeutic target for future medical intervention.

Print Article Summary Cat 2 CME Report