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See Latest ArticlesHaplotype and Cell Proliferation Analyses of Candidate Lung Cancer Susceptibility Genes on Chromosome 15q24-25.1
Cancer Research, 10/06/09
Liu Y et al. – Down–regulation of PSMA4 expression decreased proteasome activity and induced apoptosis. Proteasome dysfunction leads to many diseases including cancer, and drugs that inhibit proteasome activity show promise as a form of cancer treatment. Genes around hapN were also investigated, but did not show any direct effect on lung cancer cell proliferation. PSMA4 is a strong candidate mediator of lung cancer cell growth, and may directly affect lung cancer susceptibility through its modulation of cell proliferation and apoptosis.
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Today in Basic Science/Genetics...keeping you current
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The anti-cancer peptide, PNC-27, induces tumor cell lysis as the intact peptide
Cancer Chemotherapy and Pharmacology, 03/15/10
MicroRNAs contribute to the chemoresistance of cisplatin in tongue squamous cell carcinoma lines
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Expression of toll-like receptor 4 is down-regulated during progression of cervical neoplasia
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Today in Lung/Thoracic Oncology...keeping you current
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4beta-Hydroxywithanolide E from Physalis peruviana (golden berry) inhibits growth of human lung cancer cells through DNA damage, apoptosis and G2/M arrest
BMC Cancer, 03/15/10
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