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Differential mechanism of NF-kappaB inhibition by two glucocorticoid receptor modulators in rheumatoid arthritis synovial fibroblasts
Arthritis & Rheumatism, 11/04/09

Gossye V et al. – DEX and CpdA display a dissimilar molecular mechanism of interaction with the NF–kappaB activation pathway ex vivo. A dual pathway, partially dependent and partially independent of GR (nongenomic), may explain the gene–inhibitory effects of CpdA in RA FLS.

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