Rheumatology

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Rheumatoid arthritis and enteric bacteria
Modern Rheumatology, 06/19/09

Aoki S et al. – This review focuses on enteric bacteria as initiating or perpetuating factors in the etiopathogenesis of rheumatoid arthritis (RA). Based on the hypothesis that entrobacterial antigens that originated from intestinal flora induce rheumatoid inflammation in the joints, following topics are discussed:

• Animal models of arthritis due to Enterobacteriaceae
• Studies on humoral and cellular responses to entrobacterial antigens in RA
• Etiology of RA involving both genetic and environmental factors
• Pathogenesis of rheumatoid inflammation accompanied by joint destruction, and
• Clinical trials with basic therapeutic considerations

• Based on immunological studies on RA it was concluded that some pts with RA are sensitized to antigens common among Enterobacteriaceae
• In some pts, RA may be based on autoimmunity from molecular mimicry by entrobacterial antigens of HLA-DR4

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