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New therapeutic strategy for amyloidosis
Future Neurology, 04/27/09
Morinaga A et al. - In this study, the authors discovered that laser beam irradiation with thioflavin T could stop the extension of beta2-microglobulin fibrils and even degrade preformed amyloid fibrils. These reactions require both thioflavin T and a laser beam at 442 nm; active oxygen plays an important role. This approach may be a new therapeutic strategy for destroying fibrils in dialysis-related amyloidosis as well as other types of amyloidosis or related disorders, including Alzheimer’s disease, familial amyloid polyneuropathy, Parkinson’s disease and Huntington’s disease.
Today in Amyloidosis...keeping you current
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A case with rheumatoid arthritis and systemic reactive AA amyloidosis showing rapid regression of amyloid deposition on gastroduodenal mucosa after a combined therapy of corticosteroid and etanercept
Rheumatology International, 10/16/09
Left Ventricular Ejection Time on Echocardiography Predicts Long-Term Mortality in Light Chain Amyloidosis
Journal of the American Society of Echocardiography, 11/04/09
MEFV mutations in Egyptian patients suffering from familial Mediterranean fever: analysis of 12 gene mutations
Rheumatology International, 10/16/09
Today in Basic Science/Genetics...keeping you current
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Relationship between anti-dsDNA, anti-nucleosome and anti-alpha-actinin antibodies and markers of renal disease in patients with lupus nephritis: a prospective longitudinal study
Arthritis Research & Therapy, 10/16/09
The relationship between synovial lymphocyte aggregates and the clinical response to infliximab in rheumatoid arthritis: A prospective study
Arthritis & Rheumatism, 11/04/09
Hepatoprotective effect of TNF-alpha blockade in psoriatic arthritis: A cross-sectional study
Annals of Rheumatic Diseases, 11/09/09
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