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See Latest ArticlesUric acid: bystander or culprit in hypertension and progressive renal disease
Journal of Hypertension, 10/15/08
Mene P et al. - Hyperuricemia seems to be a cofactor in Na+-sensitive hypertension, a marker and possibly itself responsible for microvascular damage through stimulation of the renin-angiotensin system, inhibition of endothelial nitric oxide, and proliferative effects on vascular smooth muscle. As fructose-rich diets increase uric acid levels, hyperuricemia may also play a role in the metabolic syndrome, triggering insulin resistance and hypertension.
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Today in Gout and Hyperuricemia...keeping you current
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The limited validity of the criteria of the American College of Rheumatology for classifying gout patients in primary care
Annals of Rheumatic Diseases, 11/30/09
Common Polymorphisms Influencing Serum Uric Acid Levels Contribute to Susceptibility to Gout, but Not to Coronary Artery Disease
PLoS ONE - Cardiovascular Disorders, 11/10/09
Associations of a nonsynonymous variant in SLC2A9 with gouty arthritis and uric acid levels in Han Chinese and Solomon Islanders
Annals of Rheumatic Diseases, 09/11/09
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