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Reciprocal cross-talk between RANKL and interferon-[gamma]-inducible protein 10 is responsible for bone-erosive experimental arthritis
Arthritis & Rheumatism, 05/14/08
Print     Email This Article     Save in My Library   Free Abstract
Kwak HB et al. - Our findings suggest that IP-10 plays a critical role in the infiltration of CD4+ T cells and F4/80+ macrophages into inflamed joints and causes bone destruction. Our results provide the first evidence that IP-10 contributes to the recruitment of inflammatory cells and is involved in bone erosion in inflamed joints.
 

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