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Beta-Amyloid(1–42) induces a reduction in the parallel fiber responses of Purkinje cells: Possible involvement of pro-inflammatory processes
Experimental Gerontology, 10/18/07
Arbez, N., et al. - In Alzheimer’s disease there is an increased production of the toxic Beta-amyloid peptides (Abeta), especially the longer forms such as Abeta(1–42). Using the patch–clamp technique we have studied the contribution of early pro-inflammatory processes to the acute effects of 1 microM Abeta(1–42) on the parallel fiber EPSC (PF-EPSC) of Purkinje cells in cerebellar slices. Abeta(1–42) induces a decrease in the PF-EPSC amplitude
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Amyloid and the GI tract
Expert Review of Gastroenterology & Hepatology, 12/03/09
MEFV mutations in Egyptian patients suffering from familial Mediterranean fever: analysis of 12 gene mutations
Rheumatology International, 10/16/09
Classification of amyloidosis by laser microdissection and mass spectrometry based proteomic analysis in clinical biopsy specimens
Blood, 10/09/09
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Increased mRNA expression of collagen V gene in pulmonary fibrosis of systemic sclerosis
European Journal of Clinical Investigation, 12/04/09
The immunoregulatory role of vitamins A, D and E in patients with primary Sjogren's syndrome
Rheumatology, 12/01/09
Cyclic AMP response element-binding protein is implicated in IL-6 production from arthritic synovial cells
Modern Rheumatology, 12/01/09
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