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Hyperoxia Disrupts Vascular Endothelial Growth Factor-Nitric Oxide Signaling and Decreases Growth of Endothelial Colony Forming Cells from Preterm Infants
Lung Cellular and Molecular Physiology, 09/11/09
Fujinaga H et al. – Hyperoxia decreases growth and disrupts VEGF–NO signaling in human preterm ECFCs. VEGF treatment restores growth in hyperoxia by increasing NO production. And NO treatment also increases growth during hyperoxia. Exogenous VEGF or NO may protect preterm ECFCs from the adverse effects of hyperoxia and preservation of ECFC function may improve outcomes of preterm infants.
Today in Basic Science/Genetics...keeping you current
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Absence of inflammation and pneumonia during infection with non-pigmented Yersinia pestis reveals new role for the pgm locus in pathogenesis
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Pattern of Antioxidant and DNA Repair Gene Expression in Normal Airway Epithelium Associated with Lung Cancer Diagnosis
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Today in Pediatric Pulmonology...keeping you current
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Anti-inflammatory drug therapy in asthma
Paediatric Respiratory Reviews, 11/02/09
A Randomized, Double-Blind Study Examining the Comparative Efficacies and Safety of Inhaled Epinephrine and Nasal Decongestant in Hospitalized Infants With Acute Bronchiolitis
The Pediatric Infectious Disease Journal, 11/17/09
A Randomized Trial of Nebulized 3% Hypertonic Saline With Epinephrine in the Treatment of Acute Bronchiolitis in the Emergency Department
Archives of Pediatrics and Adolescent Medicine, 11/23/09
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