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Rehan VK et al. – The authors have argued that since nicotine alters the normal homeostatic epithelial–mesenchymal paracrine signaling in the developing alveolus, rather than causing totally disruptive structural changes, it offers a unique opportunity to prevent, halt, and/or reverse this process through targeted molecular manipulations.

Today in Basic Science/Genetics...keeping you current

Innate Immunity in the Therapeutic Actions of Endothelial Progenitor Cells in Pulmonary Hypertension
American Journal of Respiratory Cell and Molecular Biology, 12/11/09

Absence or Inhibition of Matrix Metalloproteinase-8 Decreases Ventilator-induced Lung Injury
American Journal of Respiratory Cell and Molecular Biology, 12/11/09

Influence of DNA repair RAD51 gene variants in overall survival of non-small cell lung cancer patients treated with first line chemotherapy
Cancer Chemotherapy and Pharmacology, 12/11/09

Today in COPD...keeping you current

Screening for Depression in Chronic Obstructive Pulmonary Disease
COPD: Journal of Chronic Obstructive Pulmonary Disease, 12/11/09

Exertional Desaturation in Patients with Chronic Obstructive Pulmonary Disease
COPD: Journal of Chronic Obstructive Pulmonary Disease, 12/11/09

Dynamic hyperinflation during daily activities: does COPD gold stage matter
Chest, 12/11/09


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