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Inhibition of mTOR Attenuates Store-operated Ca2+ Entry in Cells from Endarterectomized Tissues of Patients with Chronic Thromboembolic Pulmonary Hypertension
Lung Cellular and Molecular Physiology, 07/28/09
Ogawa A et al. – The data from this study indicate the importance of the mTOR pathway in the development of pulmonary vascular remodeling in CTEPH and suggests a potential therapeutic benefit of rapamycin (or inhibition of mTOR) in these patients.
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