Pulmonology

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p16(INK4A) inactivation mechanisms in non-small-cell lung cancer patients occupationally exposed to asbestos
Lung Cancer, 05/18/09

Andujar P et al. - This study suggests that P16/CDKN2A gene inactivation in asbestos-exposed NSCLC cases mainly occurs via deletion, a feature also found in malignant mesothelioma, a tumor independent of tobacco smoking but associated with asbestos exposure, suggesting a possible relationship with an effect of asbestos fibers.

Today in Lung/Thoracic Oncology...keeping you current

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Tumor necrosis factor inhibitors and lung disease: A paradox of efficacy and risk
Seminars in Arthritis and Rheumatism, 11/20/09

Randomized phase III trial of gemcitabine-based chemotherapy with in situ RRM1 and ERCC1 protein levels for response prediction in non-small-cell lung cancer
Journal of Clinical Oncology, 11/05/09

Is neoadjuvant chemoradiotherapy a feasible strategy for stage IIIA-N2 non-small cell lung cancer? Mature results of the randomized IFCT-0101 phase II trial
Lung Cancer, 11/03/09

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Today in Occupational Health...keeping you current

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Asthma and lower respiratory symptoms in New York State employees who responded to the World Trade Center disaster
International Archives of Occupational & Environmental Health, 11/09/09

The additive value of patch testing with patients own products at an occupational dermatology clinic
Contact Dermatitis, 10/28/09

Asbestos fiber dimensions and lung cancer mortality among workers exposed to chrysotile
Occupational and Environmental Medicine, 11/06/09

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