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Glaser S et al. - Hemosiderin-laden macrophages were observed in 48% of subjects. The mean alveolar–capillary wall thickness was significantly increased to 9.9 ± 4.2 [micro]m. The time of survival after HTX was not correlated with the incidence of pathological findings. The described vascular and interstitial pulmonary changes as well as the increased membrane thickness may cause the persistent impairment of KCO after HTX.

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