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Priming Deficiency in Male Subjects at Risk for Alcoholism: The N4 During a Lexical Decision Task
Alcoholism, 09/23/09
Porjesz B et al. – The lack of N4 attenuation to primed stimuli and/or inability to differentiate between primed and unprimed stimuli, without latency and reaction time being affected, suggest deficits in semantic priming, especially in semantic expectancy and/or postlexical semantic processing in HR male offspring. Further, it indicates that it might be an electrophysiological endophenotype that reflects genetic vulnerability to develop alcoholism.
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