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See Latest ArticlesHO-2 deletion causes endothelial cell activation marked by oxidative stress, inflammation and angiogenesis
Journal of Pharmacology and Experimental Therapeutics, 09/23/09
Bellner L et al. – This switch may be the result of reduced HO activity and the associated reduction in the cytoprotective HO products, carbon monoxide and biliverdin/bilirubin, since addition of biliverdin to HO–2(–/–) cells attenuated angiogenesis and reduced superoxide production. This transformation underscores the importance of HO–2 in the regulation of endothelial cell homeostasis.
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