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See Latest ArticlesN6-substituted cAMP Analogs Inhibit bTREK-1 K+ Channels and Stimulate Cortisol Secretion by a PKA-Independent Mechanism
Molecular Pharmacology, 09/09/09
Liu H et al. – The results indicate that 6–Bnz–cAMP and other 6–substituted cAMP analogs can inhibit bTREK–1 K+ channels and stimulate delayed increases in cortisol synthesis by AZF cells through a PKA– and Epac–independent mechanism. They also suggest that ACTH and cAMP function in these cells through a third cAMP–dependent protein.
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