Regulation and expression of IL-32 in chronic rhinosinusitis
Allergy, 05/16/2012
Soyka MB et al. – The induction of IL–32 by TNF–α, IFN–γ and Th1 cells as well as its increased expression in sinus tissues from chronic rhinosinusitis (CRS) patients with nasal polyps demonstrated a potential role for IL–32 in the pathogenesis of CRS.



