Regulation and expression of IL-32 in chronic rhinosinusitis

Allergy, 05/16/2012

The induction of IL–32 by TNF–α, IFN–γ and Th1 cells as well as its increased expression in sinus tissues from chronic rhinosinusitis (CRS) patients with nasal polyps demonstrated a potential role for IL–32 in the pathogenesis of CRS.

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