Orthopedics Articles

Orthopedics

sponsor
Become a Member Today!
Register
Email:


Password:

Remember me
Forgot your Password?
Invite Code?
Article ID

Your Article Summary

(Click the title below to leave the MDLinx Network and go to the Journal's Website)

Masi L et al. - The biochemical and clinical profile of the two unrelated patients bearing the mutation reinforces the relevant role of FGF-23 in bone metabolism and in the pathogenesis of tumoral calcinosis.

Related Articles

Tumoral calcinosis in children, a challenging and possibly underdiagnosed condition
Acta Orthopaedica Belgica, 11/17/09    Relevance Score: 69%

Tumoral Calcinosis Mimicking Recurrent Osteosarcoma
Radiology Case Reports, 10/23/09    Relevance Score: 68%

Defective O-Glycosylation due to a Novel Homozygous S129P Mutation Is Associated with Lack of Fibroblast Growth Factor 23 Secretion and Tumoral Calcinosis
Journal of Clinical Endocrinology and Metabolism, 10/29/09    Relevance Score: 66%

Familial tumoral calcinosis and hyperostosis–hyperphosphataemia syndrome are different manifestations of the same disease: novel missense mutations in GALNT3
Skeletal Radiology, 10/21/09    Relevance Score: 66%

Familial Tumoral Calcinosis: From Characterization of a Rare Phenotype to the Pathogenesis of Ectopic Calcification
Journal of Investigative Dermatology, 10/30/09    Relevance Score: 65%


Sponsor

Article Search

Keyword:

Search:

Published within

Sort By:
Date
Relevance


Sponsor

Sponsor

Send this Summary to a Colleague

Enter email address