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See Latest ArticlesThe PI3K/Akt and mTOR/P70S6K signaling pathways in human uveal melanoma cells: interaction with B-Raf/ERK
Investigative Ophthalmology and Visual Science, 08/07/09
Babchia N et al. – Activated PI3K/Akt attenuates the inhibitory effects of rapamycin on cell proliferation and thus serves as a negative feedback mechanism. This finding suggests that rapamycin is unlikely to inhibit uveal melanoma growth. In contrast, targeting PI3K while inhibiting B–Raf/ERK may be a promising approach to reduce proliferation of uveal melanoma cells.
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Melanoma-Associated Retinopathy
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