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(Click the title below to leave the MDLinx Network and go to the Journal's Website)

Shen J et al. – These data demonstrate that exogenous Epo significantly attenuates the retinal neuronal cell death induced by glyoxal–AGEs by promoting anti–apoptotic and suppressing apoptotic proteins. Epo/EpoR signaling through ERK and Akt pathways is pivotal in Epo neuroprotective mechanisms.


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