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Noordam C et al. - In vitro coincubation of human SULT2A1 and wild-type or mutant PAPSS2 proteins confirmed the inactivating nature of the mutations. These observations indicate that PAPSS2 deficiency is a monogenic adrenocortical cause of androgen excess.


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Today in Pediatric/Adolescent...keeping you current

Case Management of Cervical Cytology and Histology in the Adolescent Patient
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Today in Reproductive Endocrinology...keeping you current

Comparison of Simvastatin and Metformin in Treatment of Polycystic Ovary Syndrome: Prospective Randomized Trial
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Criteria for Polycystic Ovarian Morphology in Polycystic Ovary Syndrome as a Function of Age
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