Nursing Medical News about Insulin Leptin

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Insulin, leptin and reward
Trends in Endocrinology and Metabolism, 11/02/09

Davis JF et al. – Feeding for pleasure, or “non–homeostatic feeding”, potentially contributes to the rapid development of obesity worldwide. Obesity is associated with an imbalance of regulatory hormones which normally act to maintain stable energy balance and body weight. The adiposity hormones insulin and leptin are two such signals elevated in obesity with the capacity to dampen feeding behavior through their action on hypothalamic circuits which regulate appetite and metabolism. Recent evidence suggests that both hormones achieve this degree of regulation by inhibiting the rewarding aspects of feeding behavior, perhaps by signaling within midbrain reward circuits. This review describes the capacity of both insulin and leptin to regulate reward–related behavior.

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Correlation of the leptin:adiponectin ratio with measures of insulin resistance in non-diabetic individuals
Diabetologia - Clinical and Experimental Diabetes and Metabolism, 10/12/09 Relevance Score: 69%

Effects of bilateral ovariectomy and estrogen replacement therapy on serum leptin, sex hormone binding globulin and insulin like growth factor-I levels
Gynecological Endocrinology, 11/16/09 Relevance Score: 66%

Metabolic syndrome and risk of subsequent colorectal cancer
World Journal of Gastroenterology, 11/09/09 Relevance Score: 66%

Phosphatidyl Inositol 3-Kinase Signaling in Hypothalamic Proopiomelanocortin Neurons Contributes to the Regulation of Glucose Homeostasis
Endocrinology, 10/15/09 Relevance Score: 66%

Leptin and the metabolic syndrome in patients with myotonic dystrophy type 1
Acta Neurologica Scandinavica, 10/07/09 Relevance Score: 65%