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See Latest ArticlesMitochondrial mechanisms of cell death and neuroprotection in pediatric ischemic and traumatic brain injury
Experimental Neurology, 08/07/09
Robertson CL et al. – Based on these pathways of cell dysfunction and death, several approaches toward neuroprotection are being investigated that show promise toward clinical translation. These strategies include minimizing oxidative stress by avoiding unnecessary hyperoxia, promoting aerobic energy metabolism by repletion of NAD+ and by providing alternative oxidative fuels, e.g., ketone bodies, directly interfering with apoptotic pathways at the mitochondrial level, and pharmacologic induction of antioxidant and anti–inflammatory gene expression.
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