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The role of mitochondrial transition pore, and its modulation, in traumatic brain injury and delayed neurodegeneration after TBI
Experimental Neurology, 08/12/09

Mazzeo AT et al. – Cyclosporin A, which has been reported to block the opening of mPTP, showed a significant decrease in mitochondrial damage and intra–axonal cytoskeletal destruction thereby protecting the axonal shaft and blunting axotomy. This review addresses an important issue of mPT activation after severe head injury, its role in acute post–traumatic neurodegeneration, and the rationale for targeting the mPTP in experimental and clinical TBI studies.

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