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See Latest ArticlesActivation of the prostaglandin system in response to sleep loss in healthy humans: Potential mediator of increased spontaneous pain
Pain, 06/30/09
Haack M et al. - Spontaneous pain, including headache, muscle pain, stomach pain, generalized body pain, and physical discomfort significantly increased by 5–14 units on a 100-unit scale during TSD, compared to the sleep condition. Urinary PGE2 metabolite significantly increased by about 30% in TSD over sleep condition. TSD-induced increase in spontaneous pain, in particular headache and muscle pain, was significantly correlated with increase in PGE2 metabolite. Activation of the PGE2 system appears to be a potential mediator of increased spontaneous pain in response to insufficient sleep.
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Urate as a Predictor of the Rate of Clinical Decline in Parkinson Disease
Archives of Neurology, 10/14/09
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Today in Pain Medicine...keeping you current
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Nortriptyline and gabapentin, alone and in combination for neuropathic pain: a double-blind, randomised controlled crossover trial
The Lancet, 10/13/09
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Topiramate in migraine progression
The Journal of Headache and Pain, 10/20/09
Today in Sleep...keeping you current
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Armodafinil for Treatment of Excessive Sleepiness Associated With Shift Work Disorder: A Randomized Controlled Study
Mayo Clinic Proceedings, 11/03/09
Validation of ECG-derived sleep architecture and ventilation in sleep apnea and chronic fatigue syndrome
Sleep and Breathing, 10/15/09
Review: continuous positive airway pressure devices are effective and cost-effective for obstructive sleep apnoea
Evidence-Based Medicine, 10/22/09
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