Your Article Summary
See Latest ArticlesRET Expression and Neuron-like Differentiation of Pheochromocytoma and Normal Chromaffin Cells
Hormone and Metabolic Research, 06/29/09
Powers JF et al. - The findings suggest that overexpression of RET in pheochromocytomas could result either from a secondary event that activates signaling pathways mediating adult chromaffin cell plasticity or as a component of a persistent sympathoadrenal progenitor phenotype. Whether wild-type RET contributes to tumor development or is merely a lineage marker for cells at various stages of neuronal differentiation may vary, with other tumor characteristics.
Related Articles
Isocitrate Dehydrogenase Mutations Are Rare in Pheochromocytomas and Paragangliomas
Journal of Clinical Endocrinology and Metabolism, 11/25/09
Relevance Score: 87%
Clinical Predictors and Algorithm for the Genetic Diagnosis of Pheochromocytoma Patients
Clinical Cancer Research, 10/19/09
Relevance Score: 87%
Low Sensitivity of Glucagon Provocative Testing for Diagnosis of Pheochromocytoma
Journal of Clinical Endocrinology and Metabolism, 11/24/09
Relevance Score: 85%
ACTH-producing pheochromocytoma: Clinical considerations and concise review of the literature
European Journal of Internal Medicine, 10/12/09
Relevance Score: 85%
Radiofrequency Ablation of Metastatic Pheochromocytoma
Journal of Vascular and Interventional Radiology, 11/13/09
Relevance Score: 83%
Today in Neuroendocrinology...keeping you current
Receive free subspecialty "5-minute updates" via email
Evaluation of antioxidant systems in pituitary-adrenal axis diseases
Pituitary, 12/18/09
A heritable predisposition to pituitary tumors
Pituitary, 12/18/09
The risk for breast cancer is not evidently increased in women with hyperprolactinemia
Pituitary, 12/18/09