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See Latest ArticlesRET Expression and Neuron-like Differentiation of Pheochromocytoma and Normal Chromaffin Cells
Hormone and Metabolic Research, 06/29/09
Powers JF et al. - The findings suggest that overexpression of RET in pheochromocytomas could result either from a secondary event that activates signaling pathways mediating adult chromaffin cell plasticity or as a component of a persistent sympathoadrenal progenitor phenotype. Whether wild-type RET contributes to tumor development or is merely a lineage marker for cells at various stages of neuronal differentiation may vary, with other tumor characteristics.
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Neuroendocrine carcinomas arising in solid-organ transplant recipients: rare but aggressive malignancies
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