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See Latest ArticlesAssociation of FAK activation with lentivirus-induced disruption of blood-brain barrier tight junction-associated ZO-1 protein organization
Journal of Neurovirology, 06/12/09
Ivey NS et al. - The present data demonstrate that disruption of tight junctions between BMECs is mediated through activation of focal adhesion kinase (FAK) by phosphorylation at TYR-397. Inhibition of FAK activation is sufficient to prevent tight junction disruption. Thus, it may be possible to inhibit the development of HIVE by using inhibitors of FAK.
Today in Basic Science/Genetics...keeping you current
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Micro-ribonucleic acid profiling of multiple sclerosis lesions identifies modulators of the regulatory protein CD47
Brain, 12/04/09
Apolipoprotein E genotype and cerebral palsy
Developmental Medicine & Child Neurology, 12/04/09
Cyclooxygenase-2 (COX-2) G-765C is a protective factor for coronary artery disease but not for ischemic stroke: A meta-analysis
Atherosclerosis, 12/04/09
Today in CNS Infectious Disease...keeping you current
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Dysregulated Epstein-Barr virus infection in patients with CIDP
Journal of Neuroimmunology, 12/04/09
Limited Role of CD4+Foxp3+ Regulatory T Cells in the Control of Experimental Cerebral Malaria
Journal of Immunology, 12/04/09
Influence of Malnutrition on the Course of Childhood Bacterial Meningitis
The Pediatric Infectious Disease Journal, 11/30/09
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