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See Latest ArticlesLoss of Tsc1, but not Pten, in renal tubular cells causes polycystic kidney disease by activating mTORC1
Human Molecular Genetics, 11/03/09
Zhou J et al. – Disruption of Pten, an upstream regulator of TSC1/TSC2, in the same cells, does not lead to PKD seemingly due to limited activation of mTORC1, suggesting that PTEN may not be a major upstream regulator of TSC/mTORC1 during early postnatal kidney development.
Today in Basic Science/Genetics...keeping you current
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Simultaneous Infiltration of Polyfunctional Effector and Suppressor T Cells into Renal Cell Carcinomas
Cancer Research, 11/03/09
Mesenchymal Stem Cell Homing Capacity
Transplantation, 10/29/09
Molecular testing for adult type Alport syndrome
BMC Nephrology, 11/18/09
Today in Cystic Dz...keeping you current
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The Role of the Mammalian Target Of Rapamycin (mTOR) in Renal Disease
Journal of the American Society of Nephrology, 11/04/09
Autosomal Dominant Polycystic Kidney Disease: New Treatment Options and How to Test Their Efficacy
Kidney and Blood Pressure Research, 11/06/09
Autosomal Dominant Polycystic Kidney Disease: Current and Emerging Treatments
Kidney, 10/30/09
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