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(Click the title below to leave the MDLinx Network and go to the Journal's Website)

Ohse T et al. – The authors propose that together with its underlying Bowman's basement membrane, the tight junctions of PECs serve as a second barrier to protein. When disturbed following PEC injury, the increase in permeability of this layer to filtered protein is a mechanism underlying peri–glomerular inflammation characteristic of anti–GBM disease.

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