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Thrombospondin-2 therapy ameliorates experimental glomerulonephritis via inhibition of cell proliferation, inflammation, and TGF- activation
American Journal of Physiology: Renal Physiology, 09/03/09
Daniel C et al. – The data demonstrate TSP–2 as a potential therapeutic agent to inhibit the glomerular proliferative and inflammatory response as well as TGF–beta activation and extracellular matrix accumulation in experimental mesangial proliferative glomerulonephritis.
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