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(Click the title below to leave the MDLinx Network and go to the Journal's Website)

Yang F et al. – Ang II induces tubular CTGF expression and renal fibrosis via the TGF––dependent and –independent Smad3 signaling pathways, suggesting that targeting Smad3 may have therapeutic potential for hypertensive nephropathy.


Today in Glomerular/Tubular Dz...keeping you current

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Today in Nephropathies...keeping you current

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Renal artery stenosis: pathophysiology and treatment
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