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See Latest ArticlesGEC-targeted HO-1 Expression Reduces Proteinuria in Glomerular Immune Injury
American Journal of Physiology: Renal Physiology, 07/09/09
Duann P et al. - Induction of HO-1 is a key defense mechanism against oxidative stress. Compared to tubules, glomeruli are refractory to HO-1 upregulation in response to injury. This can be a disadvantage as it may be associated with insufficient production of cytoprotective heme degradation metabolites.
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Today in Basic Science/Genetics...keeping you current
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Paracrine/endocrine mechanism of stem cells on kidney repair: role of microvesicle-mediated transfer of genetic information
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Susceptibility genes in common complex kidney disease
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Today in Glomerular/Tubular Dz...keeping you current
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Association of High Pretransplant sIL-6R Plasma Levels with Acute Tubular Necrosis in Kidney Graft Recipients
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N-Acetylcysteine Amide Protects Renal Proximal Tubular Epithelial Cells against Iohexol-Induced Apoptosis by Blocking p38 MAPK and iNOS Signaling
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Renal homeostasis and tubuloglomerular feedback
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