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Havasi A et al. - Hsp27 over-expression prevents sublethal REC injury at cell contact sites possibly by a c-Src dependent mechanism. Further exploration of the biochemical link between Hsp27 and c-Src could yield therapeutic interventions for ameliorating ischemic renal cell injury and organ dysfunction.

Today in Basic Science/Genetics...keeping you current

Paracrine/endocrine mechanism of stem cells on kidney repair: role of microvesicle-mediated transfer of genetic information
Current Opinion in Nephrology and Hypertension, 12/17/09

Susceptibility genes in common complex kidney disease
Current Opinion in Nephrology and Hypertension, 12/17/09

The Changing Phenotype of Academic Nephrology— A Future at Risk
Clinical Journal of the American Society of Nephrology, 12/15/09