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See Latest ArticlesPore-forming toxins activate MAPK p38 by causing loss of cellular potassium
Biochemical and Biophysical Research Communications, 06/23/09
Kloft N et al. - Mitogen activated protein kinase (MAPK) p38 has emerged as a survival protein in cells that are attacked by bacterial toxins forming small membrane pores. The authors propose that PFT trigger the p38 MAPK-pathway by causing loss of cellular K+.
Today in Basic Science/Genetics...keeping you current
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Fibroblast growth factor 23 and the future of phosphorus management
Current Opinion in Nephrology and Hypertension, 10/15/09
The balance of autocrine VEGF-A and VEGF-C determines podocyte survival
American Journal of Physiology: Renal Physiology, 10/15/09
Simultaneous Infiltration of Polyfunctional Effector and Suppressor T Cells into Renal Cell Carcinomas
Cancer Research, 11/03/09
Today in Fluids and Electrolytes...keeping you current
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Systematic Review: Sodium Bicarbonate Treatment Regimens for the Prevention of Contrast-Induced Nephropathy
Annals of Internal Medicine, 11/03/09
Does Diabetes Mellitus Predispose to Increased Fluid Overload in Peritoneal Dialysis Patients
Nephron: Clinical Practice, 10/12/09
Guided optimization of fluid status in haemodialysis patients
Nephrology Dialysis Transplantation, 10/02/09
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