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See Latest ArticlesPore-forming toxins activate MAPK p38 by causing loss of cellular potassium
Biochemical and Biophysical Research Communications, 06/23/09
Kloft N et al. - Mitogen activated protein kinase (MAPK) p38 has emerged as a survival protein in cells that are attacked by bacterial toxins forming small membrane pores. The authors propose that PFT trigger the p38 MAPK-pathway by causing loss of cellular K+.
Today in Basic Science/Genetics...keeping you current
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Simultaneous Infiltration of Polyfunctional Effector and Suppressor T Cells into Renal Cell Carcinomas
Cancer Research, 11/03/09
Loss of Tsc1, but not Pten, in renal tubular cells causes polycystic kidney disease by activating mTORC1
Human Molecular Genetics, 11/03/09
Mesenchymal Stem Cell Homing Capacity
Transplantation, 10/29/09
Today in Fluids and Electrolytes...keeping you current
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Systematic Review: Sodium Bicarbonate Treatment Regimens for the Prevention of Contrast-Induced Nephropathy
Annals of Internal Medicine, 11/03/09
New aspects in the pathogenesis, prevention, and treatment of hyponatremic encephalopathy in children
Pediatric Nephrology, 11/11/09
Does Concomitant Administration of Sevelamer and Calcium Carbonate Modify the Control of Phosphatemia
Therapeutic Apheresis and Dialysis, 11/04/09
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