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See Latest ArticlesUric acid: bystander or culprit in hypertension and progressive renal disease
Journal of Hypertension, 10/15/08
Mene P et al. - Hyperuricemia seems to be a cofactor in Na+-sensitive hypertension, a marker and possibly itself responsible for microvascular damage through stimulation of the renin-angiotensin system, inhibition of endothelial nitric oxide, and proliferative effects on vascular smooth muscle. As fructose-rich diets increase uric acid levels, hyperuricemia may also play a role in the metabolic syndrome, triggering insulin resistance and hypertension.
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Today in Acid-Base...keeping you current
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Acid-base regulation of angiotensin receptors in the kidney
Current Opinion in Nephrology and Hypertension, 12/11/09
Activation of p53 Promotes Renal Injury in Acute Aristolochic Acid Nephropathy
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Autosomal dominant pseudohypoaldosteronism type 1 with a novel splice site mutation in MR gene
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Today in Hypertension...keeping you current
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Hypertension Awareness, Treatment, and Control in Adults With CKD: Results From the Chronic Renal Insufficiency Cohort (CRIC) Study
American Journal of Kidney Diseases, 12/11/09
Hypertension in the Developing World: Challenges and Opportunities
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Characterization and treatment of resistant hypertension
Current Cardiology Reports, 12/11/09
Today in Vascular...keeping you current
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Targeting bone morphogenetic protein signaling on renal and vascular diseases
Current Opinion in Nephrology and Hypertension, 12/11/09
Contributions of blood pressure to proteinuria and renal function in the puerperium
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