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Inhibition of Cystathionine-(beta)-synthase activity during renal ischemia-reperfusion: Role of pH and Nitric Oxide
American Journal of Physiology: Renal Physiology, 08/15/08
Prathapasinghe GA et al. - Results suggested that metabolic acidosis during ischemia and accumulation of NO metabolites during reperfusion contributed, in part, to reduced CBS activity leading to an elevation of renal Hcy levels, which in turn, played a detrimental role in the kidney.
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