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See Latest ArticlesHuman H+ATPase a4 subunit mutations causing renal tubular acidosis reveal a role for interaction with phosphofructokinase-1
American Journal of Physiology: Renal Physiology, 07/21/08
Su Y et al. - The R-to-Q mutation dramatically reduced a-subunit production, abolishing H+ATPase function completely. Thus in the context of dRTA, stability and function of the metabolon composed of H+ATPase and glycolytic components can be compromised by either loss of required PFK-1 binding (G820R), or loss of pump protein.
Today in Acid-Base...keeping you current
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Lactic Acidosis Associated With Metformin in a Patient With Type 2 Diabetes With No Contraindication
The Endocrinologist, 11/13/09
Acid-Base Balance in Peritoneal Dialysis Patients: A Stewart-Fencl Analysis
Renal Failure, 10/29/09
Effect of high-normal compared with low-normal arterial pH on protein balances in automated peritoneal dialysis patients
American Journal of Clinical Nutrition, 10/29/09
Today in Basic Science/Genetics...keeping you current
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Simultaneous Infiltration of Polyfunctional Effector and Suppressor T Cells into Renal Cell Carcinomas
Cancer Research, 11/03/09
Loss of Tsc1, but not Pten, in renal tubular cells causes polycystic kidney disease by activating mTORC1
Human Molecular Genetics, 11/03/09
Molecular testing for adult type Alport syndrome
BMC Nephrology, 11/18/09
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