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Chen XY et al. – The data suggest that Mifepristone may be exert its anti–implantation function by increasing NK cytotoxicity. The increasing NK cytotoxicity of mifepristone is not related to CD94/NKG2A and NKG2D. In the secretory phase down–regulated CD94/NKG2A, NKG2D, and NK cytotoxicity may benefit with embryo implantation.

   

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