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See Latest ArticlesAccelerated thymic atrophy as a result of elevated homeostatic expression of the genes encoded by the TNF/lymphotoxin cytokine locus
European Journal of Immunology, 09/28/09
Liepinsh DJ et al. – Results of backcrossing to TNFR1–, LTbetaR– or TNF/LT–deficient backgrounds and of reciprocal bone marrow transfers implicated both LTalpha–/LT–beta to LTbetaR and TNF/LT–alpha to TNFR1 signaling in accelerated thymus degeneration. The authors hypothesize that chronic infections can promote thymic atrophy by upregulating LT and TNF production.
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