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GRP78 Upregulation by Atheroprone Shear Stress via p38-, 21-Dependent Mechanism in Endothelial Cells
Arteriosclerosis, Thrombosis, and Vascular Biology, 06/16/08
Print     Email This Article     Save in My Library   Free Abstract
Feaver RE et al. - This study supports a role of the hemodynamic environment in preferentially inducing GRP78 and the UPR in atheroprone regions, before lesion development, and suggests a potential atheroprotective, compensatory effect in response to ER stress within atherosclerotic lesions.
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