Painful nerve injury increases plasma membrane Ca2+ -ATPase activity in axotomized sensory neurons Full Text
Molecular Pain, 06/21/2012
Gemes G et al. – Authors found that PMCA function is elevated in axotomized sensory neurons, which contributes to neuronal hyperexcitability. Accelerated PMCA function in the primary sensory neuron may contribute to the generation of neuropathic pain, and thus its modulation could provide a new pathway for peripheral treatment of post–traumatic neuropathic pain
