Modification of altered ankle motor control after stroke using focal application of Botulinum toxin type A
Clinical Neurology and Neurosurgery,  Clinical Article

Tang SF et al. – Botulinum toxin type A (BTX–A) is effective in reducing antagonistic and distant muscle activation that impedes volitional dorsiflexion.

Methods
  • 15 post-stroke and 10 matched neurologically intact subjects.
  • Modified Ashworth Scale (MAS) and Fugl–Meyer assessment of physical function scale scores along with surface EMG collected before and up to 12weeks after BTX-A injections to plantar flexor muscle motor points in stroke subjects.
  • Saline placebo injections were performed in a subset of stroke subject group.

Results
  • MAS scores were decreased at 4, 8 and 12weeks but F–M scores did not improve until 12weeks post injection.
  • Multi-muscle EMG patterns showed the return of volitional dorsiflexor activity in 11 and a decrease of antagonistic and distant coactivation in all but one of the 15.

Please login or register to follow this author.
Are you sure you want to Unfollow this Author?
► Click here to access PubMed, Publisher and related articles...
<< Previous Article | Next Article >>

    Currently, there are no available articles.

Your Unread Messages in Internal Medicine

See All >> Messages include industry-sponsored communications and special communications from MDLinx

Most Popular Internal Medicine Articles

Indexed Journals in Internal Medicine: New England Journal of Medicine, The Lancet, Archives of Internal Medicinemore

Register now to view all the MDLinx contents (FREE)!

  • Stay current on the latest literature, research and clinical news
  • Get special communications and offers from MDLinx and our sponsors
  • Receive invitations to paid market research
View Samples and Register

Connect with us, stay current.

Receive the latest mecial news
updates for free via email

Sign up!

Subscribe to our free RSS feeds:

Get the latest news in your specialty automatically added to your newsreader or your personal My Yahoo!, Google, My MSN or My AOL page. Learn More

Close