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See Latest ArticlesLoss of the Brm-Type SWI/SNF Chromatin Remodeling Complex Is a Strong Barrier to the Tat-Independent Transcriptional Elongation of Human Immunodeficiency Virus Type 1 Transcripts
Journal of Virology, 10/28/09
Mizutani T et al. – The authors propose that Brm is required for the occasional transcriptional elongation of the HIV–1 provirus in the absence of Tat. Since the Brm–type SWI/SNF complex is expressed at marginal levels in resting CD4+ T cells and is drastically induced upon CD4+ T–cell activation, the authors speculate that it plays crucial roles in the early Tat–independent phase of HIV transcription in affected patients.
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