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See Latest ArticlesToll-like receptor signaling in airborne Burkholderia thailandensis infection
Infection and Immunity, 10/12/09
West TE et al. – The findings suggest that redundancy in TLR signaling or other MyD88–dependent pathways may be important in pneumonic B. thailandensis infection but that MyD88–independent mechanisms of inflammation are also activated. TLR signaling in B. thailandensis infection is substantially comparable to signaling induced by virulent B. pseudomallei. These studies provide additional insights into the host–pathogen interaction in pneumonic Burkholderia infection.
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