Oncology Medical News about Glutathione S-transferase

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Glutathione S-transferase gene deletions and their effect on iron status in HbE/beta thalassemia patients
Annals of Hematology, 10/23/09

Sharma V et al. – Iron overload and oxidative stress are main pathophysiological features of HbE/eta thalassemia patients. Glutathione S–transferase genes (GSTT1 and GSTM1) are well known detoxification agents, and any mutation in the gene is known to cause oxidative damage. This is the first study to prove the role of GST gene deletions with iron overload in HbE/beta thalassemia.

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Glutathione S-transferase polymorphisms and survival in African-American and White colorectal cancer patients
Cancer Epidemiology, 10/22/09 Relevance Score: 66%

Glutathione S-transferase Catalyzed Desulfonylation of a Sulfonylfuropyridine
Drug Metabolism and Disposition, 10/05/09 Relevance Score: 66%

No evidence for glutathione S-transferases GSTA2, GSTM2, GSTO1, GSTO2, and GSTZ1 in breast cancer risk
Breast Cancer Research and Treatment, 11/04/09 Relevance Score: 65%

Polymorphism of glutathione S-transferases M1 and T1: susceptibility to solar keratoses in an Italian population
Clinical and Experimental Dermatology, 10/26/09 Relevance Score: 64%

Glutathione S-Transferase-mu1 Regulates Vascular Smooth Muscle Cell Proliferation, Migration, and Oxidative Stress
Hypertension, 10/14/09 Relevance Score: 64%