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See Latest ArticlesInduction of Prostatic Intraepithelial Neoplasia and Modulation of Androgen Receptor by ETS Variant 1/ETS-Related Protein 81
Cancer Research, 10/21/09
Shin S et al. – The study substantiates that ETV1 overexpression is an underlying cause in the development of prostate and possibly also seminal vesicle cancer. Its interaction with and activation of AR provides a molecular mechanism on how ETV1 exerts its deleterious function. Thus, inhibiting ETV1 or blocking its interaction with AR may represent novel strategies in prostate cancer therapy.
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Today in Basic Science/Genetics...keeping you current
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Decreased expression of KLF6 and its significance in gastric carcinoma
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Today in Renal/Urologic...keeping you current
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Annals of Diagnostic Pathology, 12/16/09
Ezrin mediates c-Myc actions in prostate cancer cell invasion
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