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See Latest ArticlesThe prolyl-hydroxylase EGLN3 and not EGLN1 is inactivated by methylation in plasma cell neoplasia
European Journal of Haematology, 10/14/09
Hatzimichael E et al. – The data suggest that the prolyl–hydroxylases may be a novel class of potential tumour suppressors in plasma cell neoplasia that warrant further investigation with regard to their potential utility as biomarkers. EGLN3 is also methylated at high frequency in B–cell lymphoma subtypes, implying that loss of EGLN3 is an important epigenetic event not only in plasma cell neoplasias but also in B–cell neoplasias.
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Today in Basic Science/Genetics...keeping you current
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Decreased expression of KLF6 and its significance in gastric carcinoma
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Today in Leukemia/Lymphoma...keeping you current
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A randomized, controlled, double-blind, pilot study of milk thistle for the treatment of hepatotoxicity in childhood acute lymphoblastic leukemia (ALL)
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