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See Latest ArticlesThe synergy of panobinostat plus doxorubicin in acute myeloid leukemia suggests a role for HDAC inhibitors in the control of DNA repair
Leukemia, 10/13/09
Maiso P et al. – The drug combination provoked a strong activation of a DNA damage response, indicating that this combination may trigger cell death by a mechanism that induced DNA double–strand breaks. These data indicate that the combination of panobinostat and doxorubicin may be an effective therapy for the treatment of AML.
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Today in Basic Science/Genetics...keeping you current
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Gene expression profiling identifies emerging oncogenic pathways operating in extranodal NK/T-cell lymphoma, nasal-type
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The sumoylation pathway is dysregulated in multiple myeloma and is associated with adverse patient outcome
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TCEAL7, a putative tumor suppressor gene, negatively regulates NF-kappaB pathway
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Today in Leukemia/Lymphoma...keeping you current
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Gene expression profiling identifies emerging oncogenic pathways operating in extranodal NK/T-cell lymphoma, nasal-type
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Differential subcellular localization regulates c-Cbl E3 ligase activity upon Notch3 protein in T-cell leukemia
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Today in Pharmacology/Therapy...keeping you current
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Diabetes and Cardiovascular Disease During Androgen Deprivation Therapy: Observational Study of Veterans With Prostate Cancer
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